What is the primary mechanism of action for Cefdinir?

Cefdinir functions primarily by inhibiting cell wall synthesis, which is a characteristic mechanism of action for the class of antibiotics known as cephalosporins. This specific action involves targeting the penicillin-binding proteins (PBPs) located in the bacterial cell wall. When these proteins are inhibited, it disrupts the cross-linking of peptidoglycan layers that provide structural integrity to the bacteria. As a result, the cell wall becomes weakened, leading to cell lysis and ultimately, bacterial death.

This mechanism is particularly effective against a range of Gram-positive and Gram-negative bacteria, making Cefdinir a valuable option in treating various bacterial infections. By focusing on cell wall synthesis, Cefdinir effectively stops the growth and multiplication of bacteria without affecting human cells, which do not possess cell walls. Other mechanisms listed, such as the inhibition of protein synthesis or disruption of RNA synthesis, apply to different classes of antibiotics and do not pertain to Cefdinir's mode of action.